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In a study conducted in a small cohort of subjects of American Indian or Mexican ancestry, a structural polymorphism of apoC1, the T45S variant, which is associated with a higher propensity of the protein to undergo N-terminal truncation, was found to be associated with higher prevalence of diabetes secondarily to the onset of obesity [112]

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It is proposed that HE in ACLF is clinically distinct from that in acute decompensation driven by diverse pathological mechanisms in each entity [31], underscoring the multifactorial pathogenesis of this condition

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The found: "The present observations suggest that dysregulation of miR-9 and miR-599 chronologically precedes CHF, suggesting that these miRNA warrant further investigation as putative initiating factors for CHF, while others were found only at the time of CHF (miR-181c and miR-495), which denotes a role in initiation, progression or consequence of CHF