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Prx1 and Prx2 KO mice Genetic deficiency of the cytosolic Prx1 in mice results in a severe anemic phenotype due to oxidative damage of the RBC membrane and cytosolic proteins (139)

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For example, whereas sulphide-oxidising activity is virtually undetectable in nervous system cells, colonocytes display high expression and activity of SOP enzymes, 131,132,133 consistent with the high H 2 S concentrations that result from gut microbial metabolism

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Source: Members of Antioxidant Machinery and Their Functions By Shalini Kapoor Mehta and Sivakumar Joghi Thatha Gowder