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In our study, PI3K, mTOR, Beclin-1 and LC3 II/I expression were elevated in the cerebral I/R group, along with increased levels of apoptotic makers, caspase-3, Bax, decrease of Bcl-2 content, as well as downregulation of intact neurons number in both cortex and hippocampal CA1 region

Then, NADH delivers those electrons to the mitochondria and turns back into NAD+

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By default, M e in DMSO-treated cells is equal to 0

NQO1 supports sirtuin function and its role in transcription of target genes and sensing of mitochondrial stress

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