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The model showed midbrain cholesterol accumulation, enhanced triacylglycerol (TAG) hydrolysis, accompanied by DA neuron loss, astrogliosis, increased -syn expression, and significant elevation of lipid peroxidation and ferroptosis markers, correlating with motor deficits (Maniscalchi et al., 2024)

HDL can remove cholesterol from the macrophages of the arterial wall through a number of mechanisms including an active transport mechanism

It generates a large amount of ROS, providing a solid foundation for ferroptosis to set in Gaasch et al

T.MillsN

In addition to the intrinsic compensation by Gstp3, exogenic compensation such as guidance cues and cell-cell interactions could help correct the defects, especially in orientation