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doi: 10.1186/s12866-019-1616-2 47 WoodmanseyEJMcMurdoMETMacfarlaneGTMacfarlaneS

10.3389/fmolb.2022.975582 Summary Keywords diabetic nephropathy, Acteoside, ferroptosis, Keap1/Nrf2/HO-1, AGEs/RAGE, oxidative stress, network pharmacology Citation Luo Z, Zhang S-J, Tang J-Y, Deng Y, Kang Y, Zhang X-H, Lu H-M, Wang Y-X, Liu W-J and Ma Y (2025) Integrating network analysis and experimental validation to reveal the ferroptosis-associated mechanism of acteoside in the treatment of diabetic nephropathy

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Elevated Hcy level was associated with the impaired/slow downed femoral fracture healing in mice on Hcy-supplemented diet ( n = 12) compared to mice on standard diet ( n = 13) [198]

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As a result, this hastens the degradation of the endothelial wall and the protective function of the microbiota, leading to increased accumulation of pro-inflammatory endotoxins and the prolonged spread of pathogens (Borgogna et al., 2020