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Abstract Background Targeted rapid degradation of bilirubin has the potential to thwart incipient bilirubin encephalopathy

This result is likely due to the high levels of glutamic acid and glycine, precursors of glutathione, present in CSL (Lusby et al., 1982)

CGP 48664, a new S-adenosylmethionine decarboxylase inhibitor with broad spectrum antiproliferative and antitumor activity

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