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In the presence of sufficient reducing agents, such as GSH or NADPH, aconitase can be restored to its active form

NMN administration reduced damaged lysosomes and improved autophagy, thereby reducing heart failure and extending the lifespan in p32cKO mice

Furthermore, when ROS exceeds a certain threshold, it leads to oxidative stress ( Mechanisms governing ferroptosis Ferroptosis, unlike apoptosis, autophagy, and cell necrosis, is a regulatory form of cell death that depends on iron and ROS

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Chronic dysregulation of these UPR pathways leads to enhanced cardiomyocyte apoptosis, inflammation, and structural deterioration, forming a key mechanism underlying ER dysfunction in diabetic cardiomyopathy ( 6.2.2 Ca 2+ leakage The ER functions as the main intracellular calcium reservoir