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Studies in animal models of PD, have observed BBB disruption, providing support for the vascular neurodegeneration hypothesis (Al-Bachari et al

In line with this, inhibition of lipid peroxidation by ALOX5 limits the activation of caspase 11 and pyroptosis in macrophages, which provides a potential therapy for treating sepsis [11, 126]

The study reported a concentration-related restriction of reactive oxygen species and reduced necrotic cell death

Exhaustion of mGSH is associated with mitochondrial dysfunction and accelerated cell aging

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As an essential component of glutathione peroxidase, it also helps carry out important enzyme processes that protect lipids (fats) in cell membranes