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The methylcobalamin component is generally safe in renal impairment, though accumulation of cobalt has been reported in patients with severe renal dysfunction receiving high doses of vitamin B12

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(2017) reported no difference in PCNA+ progenitor cell number 10 d after TAM in their five-month-old mice, however

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Presuming that the putative high-folatelow-vitamin B-12 interaction on serum holoTC and the metabolic markers of vitamin B-12 status is due to excess folic acid, an important consideration is at what level of exposure might these effects become metabolically and clinically relevant

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