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It reduces LPS-induced elevation of intracellular ROS levels and inhibits LPS-stimulated NOX2 mRNA and protein expression

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Proteostasis regulators have emerged as alternative approaches to circumvent CFTR defect by improving defective proteostasis thus avoiding unwanted protein-protein interactions and reinstating desirable interactions for misfolded CFTR variants

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This review explores the multifaceted roles of EVs in CF lung disease, focusing on their biogenesis, cargo, and contributions to disease progression