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p53 is associated with the two key kinases of ataxia telangiectasia and rad3-related protein-checkpoint kinase 1 pathway [45] that induce the transcription of cell cycle suppressor p21 gene and delay damaged G1 cells from entering S phase, preventing new start events at the beginning of the duplication process and slowing down the branching processes of reproduction of UV-irradiated cells in the S phase

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Conversely, m 5 C erasers, such as the TET family proteins and ALKBH1, are involved in the removal of these methylation marks, creating a dynamic equilibrium between the two opposing processes

For example, lncRNA PRNCR1 promotes the proliferation of breast cancer and inhibits apoptosis by regulating the microRNA-377/CCND2/MEK/MAPK axis, leading to resistance to tamoxifen in breast cancer 704 (Fig