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By binding to two different epitopes of CD38, these constructs facilitate the formation of C1q-activating oligomers, thereby increasing the interaction with C1q and enhancing the CDC effect [93]

GHK-CU uses higher doses (1-2mg) compared to BPC-157 (250-500mcg) and TB-500 (250-500mcg)

The conservative path is usually the smarter path

Pharmacokinetics: Cyanocobalamin is administered intranasally, orally, and parenterally, while hydroxocobalamin is administered only parenterally. Once absorbed, vitamin B12 is highly bound to transcobalamin II, a specific B-globulin carrier protein and is distributed and stored primarily in the liver as coenzyme B12. The bone marrow also stores a significant amount of the absorbed vitamin B12. This vitamin crosses the placenta and is distributed into breast milk. Enterohepatic recirculation conserves systemic stores. The half-life is about 6 days (400 days in the liver). Elimination is primarily through the bile; however, excess cyanocobalamin is excreted unchanged in the urine

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They are all chemically related to each other, but they have very different effects on the body