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The continued auto-oxidation of PLs is followed by increased curvature of membrane, thereby stimulating oxidative micellization, pore formation, and subsequent cell membrane damage [8]

PVL-treated neutrophils show degranulation and oxidative burst reactions and release pro-inflammatory substances such as interleukin (IL)6, IL-8 and tumour necrosis factor (TNF) 17,18 , which are generally thought to contribute to thrombus formation when it occurs in association with PVL- S