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High levels of TMAO are associated with the severity of fatty liver, and the mechanism may involve activation of the AMPK signaling pathway and interaction with oxidative phosphorylation pathways, thereby promoting hepatic lipid accumulation

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Therefore, the decreased activity of PP2A induced by chronic hypoxia is an important factor for -syn hyperphosphorylation and abnormal aggregation

So far, there is an absence of viable cure or way to halt its development