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Description

The reaction was modified to a high throughput 96-well plate format allowing many measurements in parallel

In line with this finding, homozygous mutation of DJ-1 has been linked to increased mitochondrial oxidative stress in human iPSC-derived DA neurons, a feature that was accompanied by accumulation of -synuclein and oxidized form of DA [42]

Of interest, treatment of aged rats with IGF-I was shown to upregulate eNOS and improve bioavailability of NO[295], and in vitro experiments in cultured endothelial cells demonstrated that IGF-I treatment reduce ROS production and upregulate eNOS[294]