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Further experimental work will be required to clarify whether (i) primary mitochondrial dysfunction due to mutations in (one or multiple) proteins relevant for mitochondrial homeostasis could lead to oxidative stress and metabolic disruptions or whether (ii) protein aggregation and metabolic imbalances cause secondary mitochondrial dysfunction thereby aggravating neuronal pathology

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En effet, comme mentionn prcdemment, cette forme amliore l'absorption travers les parois cellulaires, le rendant plus efficace par rapport la forme orale standard

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Myelinating oligodendrocytes express neurotransmitter receptors, ion channels, transporters, and gap junctions (108)