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2000 RNA sequencing platform, quantified using RSEM, upper quartile normalized, log 2 (x + 1) transformed, and z-normalized

Source Data for Figure S5[LSA-2023-02127_SdataFS5.zip] To address the functional impact of the mitochondrial reprogramming of BCAA metabolism in senescent cells, we blocked BCAA catabolism by knocking down the rate-limiting enzyme BCKDHA, followed by a treatment of decitabine or doxorubicin to induce senescence (Fig S5D)

Concentrations of d 9 -TMA were determined by stable isotope dilution LC-MS/MS

UK Studies suggest that by removing the senescent cells reduces inflammation, clears damaged tissue, and creates space for healthy cells to thrive

A., Pollina, E

Brain alanine formation as an ammonia-scavenging pathway during hyperammonemia: effects of glutamine synthetase inhibition in rats and astrocyte-neuron co-cultures