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In addition, GPX4 hydroxylation by PSAT1, activated by IFN in triple-negative breast cancer (TNBC), suppresses ferroptosis and reduces immunotherapy efficacy, whereas blocking this pathway restores ferroptosis sensitivity and enhances PD-1 antibody response (63)

268 Genetic studies further highlight the complexity of PERK signaling in cancer: deletion of PERK reduces Neu-dependent mammary tumor development and lung metastasis, underscoring its role in tumor progression