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Killer cellmediated parasite death, which we term 'microbe-programmed cell death' or 'microptosis', is caspase independent but resembles mammalian apoptosis, causing mitochondrial swelling, transmembrane potential dissipation, membrane blebbing, phosphatidylserine exposure, DNA damage and chromatin condensation

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Note the longer lag time in the lisdexamfetamine condition in most subjects but the otherwise similar curve shapes, including similarly steep rates of increasing amphetamine concentrations

Over 95% of animals remain within a patch of the standard bacterial food, E

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