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Copper excess triggers oxidative stress (by impairing antioxidant enzymes and NO bioavailability) and disrupts Ca homeostasis, leading to endothelial dysfunction, cardiac hypertrophy, and contractile impairment

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These free radicals furiously bounce around inside the mitochondria, striking and damaging the delicate mtDNA blueprints

Movement patterns and joint alignment also play a critical role

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Researchers study this pairing in exploratory work involving connective tissue, angiogenesis signaling, and soft tissue research pathways