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Inactive analogue of KL1 loses the ability to modulate reactive species To gain medicinal chemistry insight, we conducted a structureactivity relationship study using six KL1 analogues (Extended Data Fig

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In any event, TRIP12 regulates NF-B activity by limiting the steady-state OTUD5 protein level before TNF- stimulation, unlike other E3s regulating NF-B signaling such as LUBAC, TRAFs, and cIAP1, which are transiently activated to directly ubiquitylate the NF-B cascade regulators through K63- or M1-linked chains 46

However, sometimes antibiotic treatment causes an overgrowth of pathogenic bacteria, such as a toxin-producing bacterium called Clostridium difficile (C