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BBR can also treat T1DM by activating ERK1/2 to inhibit the differentiation of Th17 cells and blocking the activation of p38 MAPK and JNK to inhibit the differentiation of Th1 cells ( 5.4.3 BBR modulates inflammatory response by NF-B signaling pathway T2DM is a condition characterized by low-grade inflammation

Attenuation of staurosporine-induced apoptosis, oxidative stress, and mitochondrial dysfunction by synthetic superoxide dismutase and catalase mimetics, in cultured cortical neurons

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Distinct EV categories, for example, native EVs, bacterial OMVs, and EV-mimetics/hybrid systems, require tailored CMC protocols to address their unique risk profiles, including detoxification and residual DNA controls for OMVs, and stability/function validation for semi-synthetic platforms [37, 38]