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There is not yet a proposed mechanism for how antibiotic treatment affects tight junction expression in specific regions of the brain in these studies, but it could be through changes in metabolite composition or cytokine involvement

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Gut microbiota-derived components (e.g., LPS, peptidoglycan) and metabolites (e.g., SCFAs, TMAO) interact with hepatocytes via the portal vein, influencing glucose and lipid metabolism, immune responses, and redox balance, thereby driving MAFLD progression 118