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It plays a role in wound healing, tissue remodeling, and skin regeneration

930 TRPM2 induces mitochondrial lipid peroxidation in hepatic I/R injury by increasing the expression of ALOX12, and TRPM2 inhibitors or calcium depletion significantly mitigate ferroptosis caused by hepatic IR injury both in vitro and in vivo

Assessment of thoracic disc degeneration using dual-energy CT-based collagen maps

Key Points Nicotinamide adenine dinucleotide (NAD + ) is a coenzyme with roles in several cardiac and renal metabolic processes NAD + depletion is emerging as a major contributor to the pathogenesis of cardiac and renal disease Preclinical data suggest that NAD + repletion strategies have the potential to restore healthy renal and cardiac metabolism and physiology The mitochondrial sirtuins mediate some of the beneficial effects of NAD + supplementation NAD + supplementation can directly enhance metabolism and improve cellular redox reactions in the setting of cardiac and renal disease NAD + is also a substrate for enzymes involved in DNA damage repair and calcium signalling pathways

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PMID: 36332375 Biomed Res IntTargeting the lncRNA FGD5-AS1/miR-497-5p/PD-L1 Axis Inhibits Malignant Phenotypes in Colon Cancer (CC)