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It has been proposed that mitochondrial dysfunction and oxidative stress contribute to the incidence and progression of neurological disorders involving Alzheimers disease

Revascularization is an effective strategy for salvaging ischemic myocardium in myocardial infarction [194, 195], while restoration of blood flow to the ischemic myocardium paradoxically exacerbates cardiac tissue damage, a phenomenon known as myocardial ischemiareperfusion (I/R) injury [196, 197]

von Holzen U, Bocangel D, Pataer A, Vorburger SA, Liu Y, Lu X, Hunt KK, Swisher SG

Physiological application of methionine supplementation is dose-dependent, for instance, a low methionine diet suppresses the neurogenesis in animal models, whereas high dietary methionine has been associated with mild cognitive impairment primarily due to mitochondrial dysfunction (197)

10.1126/science.aaw9872 7 BaoW.-D.PangP.ZhouX.-T.HuF.XiongW.ChenK.et al (2021)

EGCG may prolong neuron survival and protect against retrograde apoptosis, potentially preventing neuronal damage [137]