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The data presented underscore some key components of hepatic nutrient-sensing pathways, particularly CRAT and SIRT3, in response to NQO1 transgenesis, which are responsible for attenuating diet-induced imbalance in glucose and lipid metabolism, cellular redox, oxidative stress, and mitochondrial dysfunction

Herein, we show that group 2 innate lymphoid cell (ILC2) conversion to ILC1s is closely involved in silicosis progression, which is mediated by activated fibroblasts via interleukin (IL)18

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As a positive control, ivermectin was applied at a concentration range from 50 to 1 ppm