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Glutamine deficiency in extracellular fluid exerts adverse effects on protein and amino acid metabolism in skeletal muscle of healthy, laparotomized, and septic rats

However, after cellular GSH depletion it is thought that NAPQI causes cytotoxicity by binding to cellular macromolecules, although the exact mechanism of cellular toxicity remains a subject of controversy [11] [12]

It is initiated through two principal pathways: the extrinsic (death receptor-mediated) and intrinsic (mitochondrial) pathways, which converge on the activation of executioner caspases (Fig

Chemico-Biological Interactions, 160(1), 1-40

Compound 2 was added to the solution of ( N - tert -butoxycarbonyl)-L-selenocysteine methyl ester in either methanol or phosphate buffer and incubated for 40 min, 24 h, and 120 h under N 2 gas balloon

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