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Kenneth McCall, published a paper in 2025 finding that compounded GLP-1s were linked to higher rates of adverse effects than non-compounded products between 2018 and 2024, including more abdominal pain, diarrhea, and nausea

[10] have shown that muscle-specific loss of carnitine acetyltransferase (CrAT) in mice led to an accumulation of acetyl-coenzyme A (CoA) within the mitochondria and allosteric inhibition of pyruvate dehydrogenase (PDH), which subsequently impaired glucose utilization

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This is because myelination needs energy and nutrition, and any systemic (respiratory or cardiac) or neurologic disease such as meningoencephalitis during the initial 1824 months of age can delay myelination, and the child can later catch up with myelination

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