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Using a murine model of heart failure, Xu et al 157 demonstrated that the soluble epoxide hydrolase inhibitor 1-adamantan-3-(5-(2-(2-ethylethoxy)ethoxy)pentyl)urea could effectively enhance the biological activities of EETs and thus improve pressure overload- or Ang II (angiotensin II)-induced myocardial hypertrophy and ventricular arrhythmias

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