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Despite their potential, the clinical translation of these bioactive components encounters significant challenges, including inefficient screening of core active constituents, undefined in vivo metabolic pathways, and inadequate analysis of multi-target interaction networks

doi:10.1016/j.ejps.2013.06.004 17

Moreover, the simultaneous inactivation of Nrf2 and blocking of TGF- signaling can completely reverse the inhibitory effect of AF on p38 MAPK phosphorylation

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Release of TNF-, IL-6, IL-18, IL-1, induces apoptosis of RGCs and degeneration of their axons (Sappington and Calkins, 2008