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As expected from the coupling of osteoblast-osteoclast functions, the loss of IGF-1 in the osteoblast also negatively affects osteoclast proliferation

Selective disruption of mitochondrial thiol redox state in cells and in vivo

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Overall, the findings support a growing body of evidence associating high UPF consumption with multiple adverse mental health outcomes alongside consistent disruptions in lipid metabolic pathways

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Such upregulation most frequently occurs via loss-of-function mutations in KEAP1, the E3 ubiquitin ligase that degrades NRF2