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The FSP1/CoQ10/NADPH system protects cells from ferroptosis in a manner independent of the GPX4 axis by inhibiting lipid peroxidation and ferroptosis through the NAD(P)H-dependent reduction of CoQ10 to ubiquinol (CoQH 2 ) [23]

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Additionally, disulfide compounds such as disulfiram (DSF) and fursultiamine disrupt the GPX4-HSC70 interaction, inhibiting GPX4 degradation and enhancing ferroptosis resistance [84]