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AMPK activation leads to phosphorylation of ACC1 at Ser79 and ACC2 at Ser212, thereby inhibiting ACC activity

Each design rule is supported below by comparative evidence and representative primary studies (2831)

PMID: 290 WangBLiuJWangQArhatteMCheongLYGlogowskaEet al

doi: 10.1136/bmj.m606.Erratumin:BMJ 3 WeiSQBilodeau-BertrandMLiuSAugerN

Similarly, KRAS mutations in tumor cells promote glutamine metabolism by inducing the expression of glutamate oxaloacetate transaminase (GOT), contributing to the maintenance of tumor cell proliferation and survival (53, 54)

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