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Age-associated mitochondrial DNA mutations cause metabolic remodeling that contributes to accelerated intestinal tumorigenesis

This general principle of methionine acting as a growth signal appears to be conserved across different organisms

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Compared with the OXA-treated mice, OXA and GSH-treated mice exhibited no significant difference in body weight loss, appetite reduction and diarrhea (data not presented), indicating that GSH treatment has no significant ameliorative effect on OXA-induced liver injury

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and the neural crest cell-like phenotypic state by the expression of AQP1, GFRA2, L1CAM, NGFR, SLC22A17 and TMEM176B 52