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Rather than relying on self-reported dietary compliance, any evaluation of clinical efficacy should be correlated with measurements of blood glucose and blood ketones (which can then be used to derive the GKI), as well other objective biological measurements (e.g., insulin, metabolic imaging, metabolomics), allowing for inter-study comparisons and external validity under different methodologies [102, 134, 145, 175]

Batf stabilizes Th17 cell development via impaired Stat5 recruitment of Ets1-Runx1 complexes

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