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It causes rapid glutathione oxidation and reactive oxygen species (ROS) formation in reactions between tBuOOH and hemoglobin

Moreover, treatment with a reducing agent that prevents disulfide stress, such as tris(2-carboxyethyl)phosphine (TCEP), specifically suppressed glucose deprivation-induced shGDH1 cell death (Extended Data Fig

A long-term deficiency can cause irreversible nerve damage, showing up as tingling, numbness, or even problems with balance

The fatty acids are absorbed by the adipocytes , but the glycerol and chylomicron remnants remain in the blood plasma, ultimately to be removed from the circulation by the liver

Life Sci 265:118826 Alberati D, Moreau J-L, Lengyel J, Hauser N, Mory R, Borroni E, Pinard E, Knoflach F, Schlotterbeck G, Hainzl D (2012) Glycine reuptake inhibitor RG1678: a pharmacologic characterization of an investigational agent for the treatment of schizophrenia

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