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In certain pathological conditions (such as periodontitis), high levels of methyl mercaptan (MeSH) act as a competitive false substrate for SQOR, reacting with the enzyme to form persulfide products (MeSSH) that cannot be further metabolized

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tHcy, sum of all forms of Hcy

In infected cells, the initiation of regulated necrosis, such as necroptosis, ferroptosis, and pyroptosis, rapidly eliminates infected cells to limit the viral spread and avoid harmful host pathogenesis (35)

Thus, the interplay between local superoxide levels and the concentration of the NO donor dictates whether the pharmacological outcome is protective or detrimental