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NAC is the primary option for patients with liver injury requiring pharmacological treatment ( FIGURE 5 Because of the absolute role of mitochondrial oxidative stress and dysfunction in the pathogenesis of AILI, the FDA approved the antioxidant NAC in 2004 to treat intrinsic DILI caused by excessive APAP intake

SJS lesions contained lower concentrations of granulysin compared to TEN lesions, which could explain the differences of clinical severity in these pathologically identical conditions

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These four points should work together

Indeed this finding is similar to studies of ischaemic damage in the brain which show MG-mediated protection occurs in the presence of similar cytokines that we show are elevated here in the ischaemic retina, including sICAM, INF, IL-1, KC(CXCL1), MCP-5 (CCL12), MIP2(CXCL2) and TNF 57

doi: 10.1016/s0006-2952(00)00556-6