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Emerging therapeutics for kidney fibrosis Despite the numerous progresses into the comprehension of the molecular mechanisms underlying kidney fibrosis, the therapeutic options currently available remain very limited and are mainly focused on slowing the progression of the disease rather than reversing the fibrotic process

Having delineated these imbalances as fundamental barriers to effective immune responses, the following sections will examine therapeutic strategies that seek to exploit these redox mismatches to enhance cancer treatment

First, Gln undergoes catalysis by glutaminase (GLS), generating glutamate (Glu), which contributes to the tricarboxylic acid (TCA) cycle by producing alpha-ketoglutarate (-KG), a carbon source for synthesizing lipids, glucose, and nucleotides [4]

& Parashuraman, S