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The potential anti-inflammatory mechanisms of SHFKO may be related to multiple bioactive components identified in our analysis: phellodendrine has been associated with reduced IL-6 and TNF- transcription in studies involving the IL-17/NF-B signaling axis

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Under cold conditions, FGF21 derived from BAT serves as a mediator of 3-adrenergic-dependent GDF15 gene transcription and is released in BAT, which suppresses proinflammatory gene expression and decreases TNF and CCL2 secretion in macrophages

The apo(a) component increases atherosclerosis and promotes clotting by interfering with the bodys clot-dissolving mechanisms