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Conversely, IAV infection can induce epithelial and macrophage senescence, leading to a senescence-associated secretory phenotype (SASP) that releases IL-1 and profibrotic factors (165), impeding epithelial regeneration and driving chronic lung damage

Chest 159 , e131e135 (2021)

Wei PC, Hsieh YH, Su MI (2012a) Loss of the oxidative stress sensor NPGPx compromises GRP78 chaperone activity and induces systemic disease

While further studies are needed to create solid therapeutic guidelines, present data suggests that vitamin C is effective as an additional therapy in the prevention and treatment of CAP

VALENTI L, CORRADINI E, ADAMS L A, et al

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