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Its mechanism is competitive inhibition within the NNMT catalytic pocket, blocking the methyl transfer from SAM to nicotinamide

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In research models, this makes it useful for examining how reduced NNMT activity changes intracellular nicotinamide handling, methyl donor utilization, and downstream metabolic signaling

Augments adipocyte energy expenditure and remodels lipid metabolism, with links to polyamine flux and epigenetic marks observed with NNMT knockdown [2]

People use both terms interchangeably