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However, with the depletion of GSH in liver cells, reactive metabolites gradually accumulate, resulting in cytotoxicity, which eventually leads to liver cell necrosis and acute liver failure (Figure 3)

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Marking experiments have shown that, in mutant strains with loss of adhesion auxiliary proteins, the P1 protein is chronically dispersed as a precursor in the cell membranes, however, it cannot aggregate to the apical organs or convert into mature P1 protein (9)