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By temporally mapping these alterations to a post-maturational window (P12 in SMA mice), we highlight a phase in which functionally mature INs begin to fail, likely as a consequence of SMN deficiencyinduced disruption of the neuronastrocyte GGG cycle, while offering a plausible developmental origin for the cortical dysfunction observed in SMA

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(1997) Immunologic findings in Peyronie's disease: a controlled study

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