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The Annexin A1/FPR2 pathway controls the inflammatory response and bacterial dissemination in experimental pneumococcal pneumonia

We used regularisation methods like dropout and weight decay on both the ViT and GAN parts to stop overfitting and make sure that training was stable with fewer epochs

Notably, IL-17 secreted by Th17 cells exerts high pathogenic potential, as it disrupts the BBB and recruits additional immune cells into the CNS, amplifying inflammatory injury [266]

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