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Pharmacological targeting of CHAC1-mediated processes therefore represents a rational therapeutic strategy, potentially disrupting the cycle of GSH depletion, ER stress amplification and iron-dependent peroxidation that drives neuronal loss

[DOI] [PubMed] [Google Scholar] 27.Ito Y, Onoda N, Okamoto T (2020) The revised clinical practice guidelines on the management of thyroid tumors by the Japan Associations of Endocrine Surgeons: core questions and recommendations for treatments of thyroid cancer

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Compared with earlier versions of EGFR inhibitors, AZD9291 continued to inhibit the EGFR/ERK pathway, leading to an improved response in their murine models and an increase in OS (140)

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