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Although iron metabolism abnormalities and lipid peroxidation mediated by ferroptosis are important mechanisms that regulate kidney diseases and ferroptosis-related defense mechanisms, including the system Xc-/GSH/GPX4 pathway, the mitochondrial pathway and the FSP1/CoQ 10 /NAD(P)H pathway, the specific details and crosstalk among these pathways are still not well understood

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Conclusion Taken together, the results reported in the present study demonstrated that the bioaccumulation of arsenic and its methylated metabolites was organ-specific and dose-specific

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A pathogenic mutation in -SNAP impairs membrane lipid binding by concealing a critical hydrophobic loop