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Quercetin inhibits hippocampal ferroptosis in diabetic encephalopathy by binding KEAP1 and upregulating the Nrf2/HO-1 pathway, reducing lipid peroxidation and iron deposition (Ren et al., 2025)

officinalis , showed nontoxic properties in LD50 tests and single-cell gel electrophoresis (60, 61)

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Therapeutic strategies aimed at reinforcing antioxidant pathways (such as GPX4, CoQ10, and BH4) may offer promising avenues to mitigate ferroptosis-related neurodegeneration

Researchers characterized the low binding affinity of G subunit to GIRK channels as well as a much higher binding affinity of G to the G subunit, which leads to a possibility that G is tethered to G when interacting with the GPCR-GIRK channel macromolecular complex (Wang, Whorton and MacKinnon, 2014)