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Qian L, Wang W, Zhou Y, Ma J

Yoshino H, Nakagawa-Hattori Y, Kondo T, Mizuno Y

These models were utilized to comprehensively analyze the metabolic landscape of activated alveolar macrophages, both in comparison to their healthy counterparts and to each other

The importance of these residues to the Aurora-A:CEP192 interaction in vivo is underscored by the phenotype of 11 cells where deleting CEP192 (aa 464511) prevents both centrosomal accumulation and autophosphorylation of Aurora-A (Fig

This study was aim to prepare a novel Que liposome (Que-lipo) and administrated via intratracheal (i.t.) with cigarette smoke induced COPD mice

Together with the primary Wilcoxon results, these sensitivity analyses confirm that the observed reduction in OMICmAge is robust to model specification and blood-cell adjustment