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Figure 3 ALA considerably suppresses CSD-induced inflammatory reaction As shown in Figure 4, CSD significantly induced hepatic inflammation by noticeably up-regulating NF-B-mediated inflammatory reaction, and increased the production of several key inflammatory factors such TNF-, IL-1, IL-6, and COX2 in hepatic tissues as examined by both immunohistochemical (Figures 4B,E,H) and biochemical approaches (Figures 4J,K)

Preclinical acute toxicity is low

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P53 phosphorylation at Ser15 enhances the affinity of both FOXO4 and FOXO4-DRI for p53 TAD2 explaining the preferential engagement of FOXO4-DRI with the hyperphosphorylated p53 characteristic of senescent cells

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